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CD79B — PLCG1

Pathways - manually collected, often from reviews:

• Reactome Reaction: CD79B → PLCG1 (indirect_complex) Brooks et al., J Immunol 2000, Marshall et al., J Exp Med 2000, Fujimoto et al., Immunity 2000, Baba et al., Proc Natl Acad Sci U S A 2001, Rodriguez et al., J Biol Chem 2001, Chiu et al., EMBO J 2002, Xu et al., J Immunol 2002, Coggeshall et al., Proc Natl Acad Sci U S A 1992, Sekiya et al., J Biol Chem 2004, Brooks et al., J Immunol 2004, Roifman et al., Biochem Biophys Res Commun 1992, Kim et al., Mol Cell Biol 2004, Carter et al., Proc Natl Acad Sci U S A 1991, Weng et al., J Biol Chem 1994, Roifman et al., Biochem Biophys Res Commun 1993, Chalupny et al., EMBO J 1993, Uckun et al., J Biol Chem 1993, de Weers et al., J Biol Chem 1994, Saxton et al., J Immunol 1994, Smit et al., J Biol Chem 1996, Kuwahara et al., Int Immunol 1996, Harmer et al., Mol Cell Biol 1997, Fu et al., J Biol Chem 1997, Bae et al., J Biol Chem 1998, Fu et al., Immunity 1998

Text-mined interactions from Literome

Chen et al., Blood 2008 (Calcium Signaling...) : CLL cells made to express ZAP-70, a kinase-defective ZAP-70 ( ZAP-70-KA ( 369 ) ), or a ZAP-70 unable to bind c-Cbl ( ZAP-YF ( 292 ) ) experienced greater intracellular calcium flux and had greater increases in the levels of phosphorylated p72(Syk), B-cell linker protein (BLNK), and phospholipase C-gamma , and greater activation of the Ig accessory molecule CD79b in response to treatment with anti-mu than did mock transfected CLL cells lacking ZAP-70
Venkataraman et al., Mol Immunol 1998 : Previous studies have shown that PKA-activation blocks BCR induced phospholipase Cgamma-activation and calcium mobilization