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EGF — NOS2

Text-mined interactions from Literome

Erickson et al., Urology 2001 (Cystitis, Interstitial) : Markers that changed after treatment were as follows : ( 1 ) nitric oxide synthase and cyclic guanosine monophosphate increased with oral L-arginine ; ( 2 ) ECP decreased with subcutaneous heparin ; ( 3 ) prostaglandin E ( 2 ) and kallikrein decreased after bladder distention ; ( 4 ) neutrophil chemotactic activity decreased after dimethyl sulfoxide ; ( 5 ) IL-2 inhibitor decreased after oral nifedipine ; ( 6 ) IL-2, IL-6, and IL-8 decreased after bacille Calmette-Guérin ( BCG ) vaccine ; and ( 7 ) APF and heparin binding epidermal growth factor changed to or toward normal levels after bladder distention or sacral nerve stimulation
Jiang et al., Arterioscler Thromb Vasc Biol 2002 (MAP Kinase Signaling System) : However, either PDGF or EGF markedly enhanced interleukin-1beta induced persistent NF-kappaB activation and iNOS expression but did not affect the early and transient NF-kappaB activation
Gallo et al., Biochem Biophys Res Commun 2002 (Carcinoma, Squamous Cell...) : LPS or EGF induced iNOS expression that was not altered by NOS or COX-2 inhibitors
Boissel et al., J Invest Dermatol 2004 : The neuronal nitric oxide synthase is upregulated in mouse skin repair and in response to epidermal growth factor in human HaCaT keratinocytes
Li et al., Mol Cell Biochem 2006 : NFAT3 is required for EGF induced COX-2 transcription, but neither iNOS transcription nor cell transformation in Cl 41 cells ... This study addressed a potential role of nuclear factor of activated T cells 3 ( NFAT3 ) in EGF induced COX-2 and iNOS transcription and cell transformation in mouse epidermal Cl 41 cells ... The EGF induced COX-2 transcription was blocked by knockdown of NFAT3 with NFAT3 siRNA, whereas the transcription of iNOS and cell transformation induced by EGF were not affected
Niidome et al., Eur J Pharmacol 2006 : We also investigated the effects of glutamate receptor antagonists, antioxidants and nitric oxide synthase inhibitor on EGF deprivation induced cell death
Broadbelt et al., American journal of physiology. Renal physiology 2009 (Ureteral Obstruction) : These data indicate that EGFR, NFkappaB, and STAT3 are required for human iNOS gene induction in response to pressure or EGF , indicating a similar mechanism of activation
Salzman et al., Am J Physiol 1996 : Induction of iNOS transcription ? by a combination of the cytokines interferon-gamma and IL-1 beta was inhibited by genistein, pyrrolidine dithiocarbamate, or dexamethasone and unaffected by pretreatment with ethylene glycol-bis ( beta-aminoethyl ether ) -N, N, N ', N'-tetraacetic acid, basic fibroblast growth factor (bFGF), epidermal growth factor (EGF) , or the isoflavone daidzein