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CSF3 — IL2
Text-mined interactions from Literome
Sugita et al., Am J Reprod Immunol 2003
:
The
effects of
G-CSF on
interleukin (IL)-2 and/or IL-12 induced interferon (IFN)-gamma production of magnetic cell sorting (MACS) sorted decidual lymphocytes was examined by enzyme linked immunosorbent spot forming cell assay ( ELISPOT )
Akahane et al., Arzneimittelforschung 1990
:
The enhanced
G-CSF production from NFSA cells may be
due at least to
interleukin-1 released from muroctasin stimulated macrophages
Leizer et al., Blood 1990
:
Cytokine regulation of colony stimulating factor production in cultured human synovial fibroblasts : I.
Induction of GM-CSF and
G-CSF production by
interleukin-1 and tumor necrosis factor
Jiang et al., J Int Med Res 2010
(Alzheimer Disease...) :
Levels of a7 nAChR protein were significantly increased and levels of
interleukin-1ß , tumour necrosis factor-a and nuclear factor-?B ( NF-?B ) protein were significantly decreased in the brain of APP transgenic mice in
response to
G-CSF
Wang et al., Int J Oncol 1995
:
We report that all-trans retinoic acid ( tRA ) synergizes with LPS to
enhance the production of granulocyte colony stimulating factor (
G-CSF ) in PMA treated cells, whereas the production of granulocyte-macrophage CSF,
interleukin 1-beta (IL-1-beta), and tumor necrosis factor-alpha (TNF-alpha) is minimally affected by tRA
Gopinath et al., PLoS Pathog 2013
:
Additionally,
G-CSF treatment inhibited
IL-2 mediated TH1 expansion
Dreger et al., Br J Haematol 1993
(Hodgkin Disease...) :
Granulocyte-colony stimulating factor induces increased serum levels of soluble
interleukin 2 receptors preceding engraftment in autologous bone marrow transplantation
Misago et al., Int J Hematol 1993
:
On the other hand,
IL-2 did not
induce G-CSF production in purified monocytes
Ogawa et al., Am J Hematol 1996
:
IL-2 , IL-3, IL-4, IL-5, and IL-6 were unable to
induce G-CSF production
Pajkrt et al., Blood 1997
(Endotoxemia) :
In group 1,
G-CSF significantly
enhanced the release of tumor necrosis factor (TNF),
interleukin-6 (IL-6), IL-8, IL-1 receptor antagonist (IL-1ra), and soluble TNF receptors