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IL2 — PRKAG1
Text-mined interactions from Literome
Giri et al., J Neurosci 2004
(Inflammation) :
A pharmacological activator of
AMPK , 5-amino-4-imidazole carboxamide riboside ( AICAR )
inhibited lipopolysaccharide (LPS) induced expression of proinflammatory cytokines ( tumor necrosis factor alpha,
interleukin-1beta , and interleukin-6 ) and inducible nitric oxide synthase in primary rat astrocytes, microglia, and peritoneal macrophages
Jhun et al., Biochem Biophys Res Commun 2006
:
In this study, we examined the
role of
AMPK in
interleukin (IL)-2 production in T cells ... Inhibition of
AMPK by compound C, a specific inhibitor of AMPK or small interfering RNA of AMPKalpha1
suppressed IL-2 production in Jurkat T cells and peripheral blood lymphocytes stimulated with PMA plus ionomycin ( PMA/Io ) or with monoclonal anti-CD3 plus anti-CD28 ... We then showed that
AMPK inhibition
reduced PMA/Io induced
IL-2 mRNA expression and IL-2 promoter activation ... These results suggest that
AMPK mediates
IL-2 production by regulating NF-AT and AP-1activation during T cell stimulation