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CD5 — IL2

Text-mined interactions from Literome

Gary-Gouy et al., J Immunol 2002 : We finally report that Y429 controls almost exclusively CD5 phosphorylation as well as inhibition of BCR triggered IL-2 production upon coaggregation of the two receptors
Gibson et al., J Mol Cell Immunol 1990 : Data presented here and elsewhere suggest that this CD5+ B-cell inducing activity is not mediated by IL-1, IL-2 , IL-3, IL-4, IL-5, IL-6, IFN-gamma, GM-CSF, or TNF
Nishimura et al., Eur J Immunol 1988 : Furthermore, expression of CD 5 restored the augmentation of IL 2 production by OKT 1 in response to cross linked anti-CD 3 mAb
Defrance et al., Eur J Immunol 1989 (Leukemia, Lymphocytic, Chronic, B-Cell) : Recombinant IL 1 alpha, IL 2 , IL 3, IL 5, IL 6, tumor necrosis factor (TNF)-alpha, IFN-gamma and granulocyte macrophage colony stimulating factor neither inhibit nor stimulate CD5 expression on B cells, therefore suggesting that the capacity to down-regulate CD5 expression on B cells is specific to IL 4
Ceuppens et al., J Immunol 1986 : Anti-CD5 had no functional effect by itself, but in association with solid-phase bound OKT3 it enhanced IL 2 receptor expression and IL 2 production and it initiated T cell proliferation
Vandenberghe et al., Immunology 1993 : In the presence of recombinant ( r ) interleukin-2 (IL-2) , anti-CD5- or anti-CD28- induced CD69 expression was further up-regulated, more sustained and, as previously shown, succeeded by IL-2 responsiveness