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CAV1 — NFKB1

Text-mined interactions from Literome

Deregowski et al., Biochem Pharmacol 2002 (Breast Neoplasms...) : Our results showed an NF-kappaB dependent induction of ephrin-A1 and caveolin-1 mRNAs after stimulation with TNF-alpha and IL-1beta, confirming that NF-kappaB controls target genes implied in tumor angiogenesis and cell transformation
Wang et al., Am J Respir Cell Mol Biol 2006 (Inflammation) : LPS induced activation of NF-kappaB and AP-1 determined by electrophoretic mobility shift assay were significantly reduced by overexpressing caveolin-1 in RAW264.7 cells
Tiruppathi et al., J Biol Chem 2008 : Role of NF-kappaB dependent caveolin-1 expression in the mechanism of increased endothelial permeability induced by lipopolysaccharide ... Thus, LPS mediates NF-kappaB dependent Cav-1 expression that results in increased caveolae number and thereby contributes to the mechanism of increased transendothelial albumin permeability
Oakley et al., J Biol Chem 2009 : Lipid rafts and caveolin-1 coordinate interleukin-1beta (IL-1beta) dependent activation of NFkappaB by controlling endocytosis of Nox2 and IL-1beta receptor 1 from the plasma membrane
Lv et al., Inflamm Res 2010 (Inflammation) : Over-expression of caveolin-1 not only increased the production of pro-inflammatory cytokine TNF-alpha and IL-6, but also enhanced the expression of the cPLA2, p38 MAPK, and NF-kappaB