◀ Back to CALM3

CALM3 — PRKAA1

Text-mined interactions from Literome

Thornton et al., J Biol Chem 2008 : Rather than restoring mRNA levels to baseline, pharmacological inhibition of Ca ( 2+ ) /calmodulin dependent protein kinase kinase beta or AMPK greatly increases Agouti related peptide and melanin concentrating hormone mRNA expression
Murgia et al., J Physiol 2009 : The Ca ( 2+ ) -calmodulin dependent kinase II ( CaMKII ), calcineurin and AMPK pathways have been implicated in GLUT4 gene regulation based on pharmacological evidence
Chikahisa et al., Neuropharmacology 2009 (Sleep Deprivation) : Sleep deprivation ( SD ) for 6 h increased p-AMPK protein in the hypothalamus and also increased the mRNA level of Ca ( 2+ ) /calmodulin (CaM) dependent protein kinase kinase beta ( CaMKK2 ), an activator of AMPK , and carnitine palmitoyltransferase 1 (CPT1), a downstream signaling factor of AMPK
Gusarova et al., Mol Cell Biol 2011 (Anoxia) : This increase in intracellular Ca ( 2+ ) induces Ca ( 2+ ) /calmodulin dependent kinase kinase ß ( CaMKKß ) -mediated AMPK activation and Na, K-ATPase downregulation
Douillard et al., Clin Exp Pharmacol Physiol 2012 (Hypertrophy) : In addition to other pathways, the Akt/mammalian target of rapamycin (mTOR) pathway, controlling protein synthesis, and the calcium/calmodulin dependent protein kinase 2 ( CamK2 ) and AMP activated protein kinase (AMPK) pathways, recently identified as calpain substrates, could be relevant in ß ( 2 ) -adrenoceptor agonist induced skeletal muscle remodelling
Brock et al., J Biol Chem 2012 (Carcinoma, Non-Small-Cell Lung...) : Our data further indicate that MIF and d-DT, acting through their shared cell surface receptor, CD74, antagonize NSCLC AMPK activation by maintaining glucose uptake, ATP production, and redox balance, resulting in reduced Ca ( 2+ ) /calmodulin dependent kinase kinase ß-dependent AMPK activation
Quan et al., Biochem Pharmacol 2013 (Fatty Liver) : Ca(+2)-calmodulin dependent protein kinase kinase ( CAMKK ) and AMP activated protein kinase (AMPK) were both activated by BA treatment
Hawley et al., J Biol Chem 1995 : The availability of alternative substrates for AMPKK and CaMKIK allowed the unequivocal demonstration that AMP and Ca2+/calmodulin promote the activation of AMPK and Ca2+/calmodulin promote the activation of AMPK and CaMKI, respectively, via three independent mechanisms : 1 ) direct activation of AMPK and CaMKI, 2 ) activation of AMPKK and CaMKIK, and 3 ) by binding to AMPK and CaMKI, inducing exposure of their phosphorylation sites