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CRK — STAT3

Text-mined interactions from Literome

Turkson et al., Mol Cell Biol 1999 (Cell Transformation, Neoplastic...) : Furthermore, inhibition of p38 and JNK activities suppresses constitutive Stat3 serine phosphorylation and Stat3 mediated gene regulation
Kakisis et al., Int J Biochem Cell Biol 2005 : Pretreating the cells with SB202190, a specific inhibitor of p38 , resulted in an increase in basal phosphorylation of ERK1/2 and a subsequent increase in basal serine phosphorylation of STAT-3
Shan et al., J Immunol 2010 (Respiratory Hypersensitivity) : Furthermore, using biochemical and genetic approaches, we found that TSLP induced proinflammatory gene expression in HASM involved the transcriptional mechanisms, MAPKs ( ERK1/2, p38 , and JNK ), and STAT3 activation
Sims et al., PloS one 2013 : Imatinib reverses doxorubicin resistance by affecting activation of STAT3 dependent NF-?B and HSP27/p38/AKT pathways and by inhibiting ABCB1
Andrés et al., Exp Dermatol 2013 (MAP Kinase Signaling System...) : TNFa, 12-O-Tetradecanoylphorbol 13-acetate ( TPA ) and UVB irradiation induced Ser727 phosphorylation of STAT3 in an ERK1/2- and p38 MAPK dependent manner, which resulted in a modulatory effect on STAT3 transcriptional activity