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CXCL12 — TNFSF10
Text-mined interactions from Literome
Lum et al., J Immunol 2005
:
Thus,
SDF-1 ligation of CXCR4 causes the parallel processes of chemotaxis and
enhanced TRAIL and TRAIL death receptor expression, resulting in apoptosis of senescent neutrophils upon their return to the bone marrow
Akimoto et al., Stem Cells Dev 2013
(Brain Neoplasms...) :
Importantly,
CXCL12 inhibited
TRAIL activation of the apoptotic pathway in GBM, suggesting that AT-MSCs may support GBM development in vivo by at least two distinct mechanisms promoting angiogenesis and inhibiting apoptosis