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Liu et al., Mol Cell Endocrinol 1999
(Adrenal Gland Neoplasms) :
Also, CBP potentiated the transcriptional activity of GAL4-SF-1 in the presence of PKA Aesøy et al., Endocrinology 2002
:
These findings were confirmed by Western blot analysis, showing that stimulation of PKA increased SF-1 protein levels ... The PKA- induced expression of SF-1 protein was not accompanied by an increase in SF-1 mRNA levels
Lund et al., Endocr Res 2002
:
Thus, whereas p/CIP stimulated SF-1 dependent transcription is further potentiated by PKA , we show that activation of PKA leads to selective downregulation of TIF2 protein and a subsequent repression of TIF2 coactivator function
Liu et al., Chin Med J (Engl) 2004
:
Activation of PKA causes wild type, but not mutant SF-1 to alter its subnuclear distribution pattern to a transactivationally active form ( foci formation )