ID:IRK9_HUMAN DESCRIPTION: RecName: Full=G protein-activated inward rectifier potassium channel 3; Short=GIRK-3; AltName: Full=Inward rectifier K(+) channel Kir3.3; AltName: Full=Potassium channel, inwardly rectifying subfamily J member 9; FUNCTION: This receptor is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium (By similarity). SUBUNIT: Associates with GIRK1 to form a G-protein-activated heteromultimer pore-forming unit (By similarity). SUBCELLULAR LOCATION: Membrane; Multi-pass membrane protein. SIMILARITY: Belongs to the inward rectifier-type potassium channel (TC 1.A.2.1) family. KCNJ9 subfamily.
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
SCOP Domains: 81296 - E set domains 81324 - Voltage-gated potassium channels
ModBase Predicted Comparative 3D Structure on Q92806
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.
Biological Process: GO:0006811 ion transport GO:0006813 potassium ion transport GO:0010107 potassium ion import GO:0034765 regulation of ion transmembrane transport