ID:EPN4_HUMAN DESCRIPTION: RecName: Full=Clathrin interactor 1; AltName: Full=Clathrin-interacting protein localized in the trans-Golgi region; Short=Clint; AltName: Full=Enthoprotin; AltName: Full=Epsin-4; AltName: Full=Epsin-related protein; Short=EpsinR; FUNCTION: Binds to membranes enriched in phosphatidylinositol 4,5- bisphosphate (PtdIns(4,5)P2). May have a role in transport via clathrin-coated vesicles from the trans-Golgi network to endosomes. Stimulates clathrin assembly. SUBUNIT: Binds clathrin heavy chain, GGA2, AP-2 and AP1G1. INTERACTION: Q9H0R8:GABARAPL1; NbExp=2; IntAct=EBI-1171113, EBI-746969; Q9GZQ8:MAP1LC3B; NbExp=2; IntAct=EBI-1171113, EBI-373144; SUBCELLULAR LOCATION: Cytoplasm. Cytoplasm, perinuclear region. Membrane; Peripheral membrane protein. Cytoplasmic vesicle, clathrin-coated vesicle. Note=Found throughout the cell, with the exception of the cell surface. Concentrated in the perinuclear region and associated with clathrin-coated vesicles close to the trans-Golgi network. TISSUE SPECIFICITY: Ubiquitously expressed at low to intermediate levels. POLYMORPHISM: Genetic variations in CLINT1 may contribute to susceptibility to schizophrenia and psychotic disorders in some populations. SIMILARITY: Belongs to the epsin family. SIMILARITY: Contains 1 ENTH (epsin N-terminal homology) domain. SEQUENCE CAUTION: Sequence=BAA11488.2; Type=Erroneous initiation;
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
ModBase Predicted Comparative 3D Structure on Q14677
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.