ID:AMOT_HUMAN DESCRIPTION: RecName: Full=Angiomotin; FUNCTION: Plays a central role in tight junction maintenance via the complex formed with ARHGAP17, which acts by regulating the uptake of polarity proteins at tight junctions. Appears to regulate endothelial cell migration and tube formation. May also play a role in the assembly of endothelial cell-cell junctions. SUBUNIT: Component of a complex whose core is composed of ARHGAP17, AMOT, MPP5/PALS1, INADL/PATJ and PARD3/PAR3. Interacts with MAGI1. Isoform 1 interacts with angiostatin. INTERACTION: Q68EM7:ARHGAP17; NbExp=4; IntAct=EBI-3891843, EBI-1642807; P35240:NF2; NbExp=2; IntAct=EBI-3903812, EBI-1014472; SUBCELLULAR LOCATION: Cell junction, tight junction. Note=Localized on the cell surface. May act as a transmembrane protein. TISSUE SPECIFICITY: Expressed in placenta and skeletal muscle. Found in the endothelial cells of capillaries as well as larger vessels of the placenta. DOMAIN: The coiled coil domain interacts directly with the BAR domain of ARHGAP17. DOMAIN: The angiostatin binding domain (871-1005) allows the binding to angiostatin. PTM: Phosphorylated upon DNA damage, probably by ATM or ATR. PTM: Polyubiquitinated by NEDD4, NEDD4L and ITCH, leading to proteasomal degradation. MISCELLANEOUS: 'Motus' means 'motility' in Latin. SIMILARITY: Belongs to the angiomotin family. SEQUENCE CAUTION: Sequence=AAH94712.1; Type=Miscellaneous discrepancy; Note=Contaminating sequence. Potential poly-A sequence;
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
ModBase Predicted Comparative 3D Structure on Q4VCS5
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.