We have a suspicion that you are an automated web bot software, not a real user. To keep our site fast for other users, we have slowed down this page. The slowdown will gradually disappear. If you think this is a mistake, please contact us at genome-www@soe.ucsc.edu. Also note that all data for hgGeneGraph can be obtained through our public MySQL server and all our software source code is available and can be installed locally onto your own computer. If you are unsure how to use these resources, do not hesitate to contact us.
UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

◀ Back to CASP16

CASP16 — NPS

Text-mined interactions from Literome

Li et al., Toxicol Lett 2010 : TiO ( 2 ) NPs effectively activated caspase-3 and -9, decreased the Bcl-2 the levels of gene and protein, and increase the levels of Bax, and cytochrome c genes and their protein expression, promoted ROS accumulation
Chung et al., J Control Release 2012 : Similar to gemcitabine HCl, GemC18-NPs induced apoptosis through caspase activation
Han et al., Biomaterials 2012 : Molecular basis of apoptosis studied by western blotting revealed that trolox-CS NPs may block mitochondria mediated apoptosis pathway through up-regulation of Bcl-2 and down-regulation of Bax and inhibiting the activation of pro-caspase 3, PARP and Bid
Yang et al., Int J Nanomedicine 2013 : In addition, the PTX-NPs markedly inhibited interleukin-6 secretion, increased caspase-8, caspase-9, and caspase-3 expression, and induced apoptosis of tumor cells in the treated mice
Song et al., Wei Sheng Yan Jiu 2013 : ZnO-NPs ( 20 microg/ml ) treatment induces early increase in caspase-1 activity followed by the increase in LDH activity and IL-1 levels, indicative of pyroptosis in A549 cells