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CASP8 — HUNK

Text-mined interactions from Literome

Sol et al., J Virol 1999 (Parvoviridae Infections) : Since B19- and NS-1 induced apoptosis was inhibited by caspase 3, 6, and 8 inhibitors, and substantial caspase 3, 6, and 8 activities were induced by NS-1 expression, there may have been interactions between NS-1 and the apoptotic pathways of the death receptors tumor necrosis factor receptor 1 and Fas
Shen et al., Adv Cancer Res 2001 (Neoplasms) : p53 mediated apoptosis can be blocked at multiple death checkpoints, by inhibiting p53 activity directly, by Bcl-2 family members regulating mitochondrial function, by E1B 19K blocking caspase-9 activation, and by caspase inhibitors
Han et al., Mol Cell Biol 1998 : Thus, E1B 19K functions through interacting with CED-4, and presumably a mammalian homologue of CED-4, to inhibit caspase activation and apoptosis