Pathways - manually collected, often from reviews:
OpenBEL Selventa BEL large corpus:
ITGA4
→
GCM1
(increases, ITGA4 Activity)
Evidence: Reporter Studies and Promoter Analysis Verify Integrin-4 and Rb1 as Primary Targets of GCMa—Overexpression of GCM1 and deletion constructs made deleting canonical GCM1 binding sites.