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PLG — VEGFA

Text-mined interactions from Literome

Chae et al., Arterioscler Thromb Vasc Biol 2000 (Disease Models, Animal) : Ang1 or VEGF induced migration and sprouting activity, increased plasmin and matrix metalloproteinase-2 secretion, and decreased tissue inhibitors of metalloproteinase type 2 secretion
Etoh et al., Cancer Res 2001 (Neovascularization, Pathologic...) : MMP-1, MMP-9, and urokinase-type plasminogen activator in ECs were strongly up-regulated by Ang-2 in the presence of VEGF in vitro
Forbes et al., Prostate Cancer Prostatic Dis 2003 (Prostatic Neoplasms) : We assessed the relative levels of secreted matrix metalloproteinases ( MMPs ) and plasminogen activators (PAs) in PC-3 cells, prostate fibroblasts and osteoblasts in the presence and absence of VEGF , TGF beta1 and bFGF
Santra et al., J Neurochem 2008 : Induction of gelatinases ( matrix metalloproteinase 2 and 9 ), the serine protease tissue plasminogen activator and plasmin by DCN transfection in MCE cells leads to extracellular proteolysis and to release of matrix bound VEGF and activation of angiogenesis
Vollmer et al., Hepatology 2009 : In addition, we show that the OSM induced HIF1alpha gene transcription and the resulting enhanced HIF1alpha protein levels are important for the OSM dependent vascular endothelial growth factor and plasminogen activator inhibitor 1 gene induction associated with several diseases