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FOS — KCNH4

Text-mined interactions from Literome

Bruning et al., Proc Natl Acad Sci U S A 2000 : Thus Rsk-2 is required for growth factor stimulated expression of c-Fos and transcriptional activation of Elk-1 and the serum response factor, but not for activation of CREB or the mitogen activated protein kinase pathway in response to PDGF and IGF-1 stimulation
Wiren et al., J Mol Endocrinol 2004 (MAP Kinase Signaling System) : Analysis of phosphorylation cascades demonstrated that DHT treatment inhibited phosphoERK1/2 levels, MAP kinase activation of Elk-1 , Elk-1 protein and phosphoElk-1 levels, and downstream AP-1/luciferase reporter activity
Yan et al., Braz J Med Biol Res 2006 : However, Western blots showed that the induction of Elk-1 regulated expression of endogenous c-Fos was not affected by Pim-1
Müller et al., J Cell Biochem 2010 (Insulinoma) : Expression of dominant negative mutants of c-Jun and Elk-1 reduced AP-1 transcriptional activity in INS-1 cells indicating that c-Jun and ternary complex factors are involved in the regulation of AP-1 activity in glucose stimulated insulinoma cells
Thiel et al., Endocrinology 2012 : The expression of a dominant negative mutant of Elk-1 reduced c-Fos expression and prevented the up-regulation of AP-1 activity as a result of calcium sensing receptor stimulation, indicating that ternary complex factors control both Egr-1- and AP-1 regulated transcription