ID:TP4A3_HUMAN DESCRIPTION: RecName: Full=Protein tyrosine phosphatase type IVA 3; EC=3.1.3.48; AltName: Full=PRL-R; AltName: Full=Protein-tyrosine phosphatase 4a3; AltName: Full=Protein-tyrosine phosphatase of regenerating liver 3; Short=PRL-3; Flags: Precursor; FUNCTION: Protein tyrosine phosphatase which stimulates progression from G1 into S phase during mitosis. Enhances cell proliferation, cell motility and invasive activity, and promotes cancer metastasis. May be involved in the progression of cardiac hypertrophy by inhibiting intracellular calcium mobilization in response to angiotensin II. CATALYTIC ACTIVITY: Protein tyrosine phosphate + H(2)O = protein tyrosine + phosphate. ENZYME REGULATION: Inhibited by sodium orthovanadate and peroxovanadium compounds, and by pentamidine. SUBUNIT: Interacts with tubulin. SUBCELLULAR LOCATION: Cell membrane. Early endosome. TISSUE SPECIFICITY: Mainly expressed in cardiomyocytes and skeletal muscle; also found in pancreas. Consistently overexpressed in colon cancer metastasis. PTM: Farnesylated. Farnesylation is required for membrane targeting (By similarity). SIMILARITY: Belongs to the protein-tyrosine phosphatase family. SIMILARITY: Contains 1 tyrosine-protein phosphatase domain.
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
ModBase Predicted Comparative 3D Structure on O75365
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.