ID:COIL_HUMAN DESCRIPTION: RecName: Full=Coilin; AltName: Full=p80; FUNCTION: Is a component of the nuclear coiled bodies (CBS) which are involved in the function or assembly/disassembly of nucleoplasmic snRNPs. During mitosis, CBS disassemble, coinciding with a mitotic-specific phosphorylation of p80 coilin. SUBUNIT: Interacts with ANKS1B. INTERACTION: Self; NbExp=3; IntAct=EBI-945751, EBI-945751; P54253:ATXN1; NbExp=3; IntAct=EBI-945751, EBI-930964; P54253-1:ATXN1; NbExp=6; IntAct=EBI-945751, EBI-930975; Q8NHQ1:CEP70; NbExp=3; IntAct=EBI-945751, EBI-739624; Q8TBB1:LNX1; NbExp=2; IntAct=EBI-945751, EBI-739832; Q9NRD5:PICK1; NbExp=3; IntAct=EBI-945751, EBI-79165; P61289:PSME3; NbExp=3; IntAct=EBI-945751, EBI-355546; Q9Y3D8:TAF9; NbExp=5; IntAct=EBI-945751, EBI-2896123; Q99986:VRK1; NbExp=9; IntAct=EBI-945751, EBI-1769146; SUBCELLULAR LOCATION: Nucleus. Note=Nuclear coiled body located in the interchromatin space between the nucleolus and the nucleus. TISSUE SPECIFICITY: Found in all the cell types examined. DOMAIN: The atypical Tudor domain at the C-terminus contains two large unstructured loops, and doesn't bind methylated residues. PTM: Symmetrical dimethylation of arginine residues within the RG repeat region modulates affinity for SMN, and thus, localization of SMN complexes to the nuclear coiled bodies. SIMILARITY: Belongs to the coilin family. SIMILARITY: Contains 1 Tudor domain.
The RNAfold program from the Vienna RNA Package is used to perform the secondary structure predictions and folding calculations. The estimated folding energy is in kcal/mol. The more negative the energy, the more secondary structure the RNA is likely to have.
ModBase Predicted Comparative 3D Structure on P38432
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Orthologous Genes in Other Species
Orthologies between human, mouse, and rat are computed by taking the best BLASTP hit, and filtering out non-syntenic hits. For more distant species reciprocal-best BLASTP hits are used. Note that the absence of an ortholog in the table below may reflect incomplete annotations in the other species rather than a true absence of the orthologous gene.